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Patients receiving LASIX should be advised that they may experience symptoms from excessive fluid and/or electrolyte lossesThe postural hypotension that sometimes occurs can usually be managed by getting up slowlyPotassium supplements and/or dietary measures may be needed to control or avoid hypokalemia.
Efficacy is diminished and risk of ototoxicity increased in patients with hypoproteinemiaassociated with nephrotic syndromeototoxicity is associated with rapid injectionsevere renal impairmentuse of higher than recommended dosesconcomitant therapy with aminoglycoside antibioticsethacrynic acidor other ototoxic drugs.
Investigations into the mode of action of LASIX have utilized micropuncture studies in ratsstop flow experiments in dogs and various clearance studies in both humans and experimental animalsIt has been demonstrated that LASIX inhibits primarily the absorption of sodium and chloride not only in the proximal and distal tubules but also in the loop of HenleThe high degree of efficacy is largely due to the unique site of actionThe action on the distal tubule is independent of any inhibitory effect on carbonic anhydrase and aldosterone.
Orthostatic hypotension may occur and be aggravated by alcoholbarbiturates or narcoticsIncrease in cholesterol and triglyceride serum levels.
If you also take sucralfatetake your furosemide dose 2 hours before or 2 hours after you take sucralfate.
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